Skin Healing Lab | PIRL Experimental

PIRL Experimental · Interactive life science

How does a cut heal?

Follow the repair. Change one condition. Look for evidence.

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1. A bleeding cut

Model time · 0.0 s

Small blood vessels are damaged. Blood cells and plasma escape into the gap. Press Play to begin.

Red blood cellWhite blood cellPlateletFibrin · protein meshPlasma · fluid around blood cellsOxygen · O₂Nutrients · NWater · H₂O
  1. 1 · Bleeding
  2. 2 · Clot forms
  3. 3 · Protective cover
  4. 4 · Cells divide & move
  5. 5 · Surface closes
  6. 6 · Healed surface
0%Gap closed
WaitingCell activity
SupportedRepair conditions

Time is compressed into animation seconds. Sizes, cell numbers, and rates are illustrative; they do not predict real healing time.

Ready. Begin with the default conditions, then try changing one factor.

Compare the repair

Solid orange: your trial. Dashed navy: the default trial at the same model time. Playback speed changes viewing speed only.

Your trial: 0% closed · Default: 0% closed.

Try a controlled comparison

Each preset restarts the model. Watch a default trial, then change just one condition.

  1. Predict what will change and why.
  2. Pause at the same model time in both trials.
  3. Compare the gap, blood delivery, and newly formed cells.
  4. Explain your result using evidence from the model.
Record observations & download results

Capture the current state, change one factor, and compare. Records stay here until you clear them or close this page.

Trial / timeGap closedConditionsScab picks
No observations yet.

CSV includes the settings, stage, and model time for each observation. No student information is collected.

What this model shows—and what it simplifies
  • Closing is not the same as clotting. Platelets and fibrin help form a clot. The clot does not turn into skin. New tissue forms beneath the cover.
  • Cells divide and move. The highlighted edge cells make new cells; skin cells also migrate over the wound. Cells in deeper tissue help lay down and reorganize supporting material.
  • Blood supports the repair. The vessel is enlarged and moved close to the action to make delivery visible. The epidermis itself has no blood vessels; materials reach it from underlying tissue.
  • The stages overlap. The six labels organize the animation. Clotting, immune activity, growth, and remodeling do not wait in a perfect queue in a real wound.
  • More is not always better. Supply sliders plateau at sufficiency. Extreme zero settings are hypothetical teaching conditions; the model does not simulate tissue death, medical treatment, or whole-body illness.
  • The final frame shows a healed surface. Deeper remodeling takes longer, and repaired tissue may retain a scar. This model does not regrow severed nerves or promise restoration of the original strength.
  • Picking is a setback. The button removes the cover and damages some new tissue in this model. Changing the infection menu changes an experimental assumption; it is not a treatment action.

This is a qualitative learning model, not medical advice. The effects are chosen to make relationships visible, not calculated from a clinical dataset.

Science references: Cedars-Sinai: How wounds heal; American Academy of Dermatology: Wound care and scarring; MedlinePlus: How wounds heal.

See the drawings that inspired the simulation

Your eye can follow the same idea through different forms: an original tissue sketch, a digital cross-section, and drawings of the healing sequence. These supplied illustrations are the starting materials; the live model adds movement and refines the science.